primary human pulmonary microvascular ec Search Results


human pulmonary microvascular endothelial cells  (PromoCell)
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PromoCell human pulmonary microvascular endothelial cells
Human Pulmonary Microvascular Endothelial Cells, supplied by PromoCell, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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human pulmonary macrovascular (hpaec) microvascular (hlmvec) ec  (Lonza)
90
Lonza human pulmonary macrovascular (hpaec) microvascular (hlmvec) ec
Effects of atrial natriuretic peptide (ANP) on endothelial cell (EC) barrier dysfunction induced by inflammatory agonists. EC were plated on gold microelectrodes and grown to confluence. At the time point indicated by the 1st arrow, cells were pretreated with ANP (100 nM, 20 min). At the time point indicated by the 2nd arrow, cells were stimulated with LPS (200 ng/ml; A and B), TNF-α (20 ng/ml; C), or a combination of IL-6 (25 ng/ml) and its soluble coreceptor, IL-6+SR (100 ng/ml; D), and transendothelial electrical resistance was monitored over time. Data are expressed as means ± SD of 3–10 independent experiments; n = 3 per condition for each experiment. HPAEC, human pulmonary macrovascular EC; <t>HLMVEC,</t> human lung <t>microvascular</t> EC.
Human Pulmonary Macrovascular (Hpaec) Microvascular (Hlmvec) Ec, supplied by Lonza, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Effects of atrial natriuretic peptide (ANP) on endothelial cell (EC) barrier dysfunction induced by inflammatory agonists. EC were plated on gold microelectrodes and grown to confluence. At the time point indicated by the 1st arrow, cells were pretreated with ANP (100 nM, 20 min). At the time point indicated by the 2nd arrow, cells were stimulated with LPS (200 ng/ml; A and B), TNF-α (20 ng/ml; C), or a combination of IL-6 (25 ng/ml) and its soluble coreceptor, IL-6+SR (100 ng/ml; D), and transendothelial electrical resistance was monitored over time. Data are expressed as means ± SD of 3–10 independent experiments; n = 3 per condition for each experiment. HPAEC, human pulmonary macrovascular EC; HLMVEC, human lung microvascular EC.

Journal: American Journal of Physiology - Lung Cellular and Molecular Physiology

Article Title: Atrial natriuretic peptide attenuates LPS-induced lung vascular leak: role of PAK1

doi: 10.1152/ajplung.00202.2009

Figure Lengend Snippet: Effects of atrial natriuretic peptide (ANP) on endothelial cell (EC) barrier dysfunction induced by inflammatory agonists. EC were plated on gold microelectrodes and grown to confluence. At the time point indicated by the 1st arrow, cells were pretreated with ANP (100 nM, 20 min). At the time point indicated by the 2nd arrow, cells were stimulated with LPS (200 ng/ml; A and B), TNF-α (20 ng/ml; C), or a combination of IL-6 (25 ng/ml) and its soluble coreceptor, IL-6+SR (100 ng/ml; D), and transendothelial electrical resistance was monitored over time. Data are expressed as means ± SD of 3–10 independent experiments; n = 3 per condition for each experiment. HPAEC, human pulmonary macrovascular EC; HLMVEC, human lung microvascular EC.

Article Snippet: Human pulmonary macrovascular (HPAEC) and microvascular (HLMVEC) EC were obtained from Lonza (Allendale, NJ).

Techniques: